tag:blogger.com,1999:blog-1629175743855013102.post3915595301454580521..comments2024-02-25T02:24:14.972-08:00Comments on Whole Health Source: Low Micronutrient Intake may Contribute to ObesityStephan Guyenethttp://www.blogger.com/profile/09218114625524777250noreply@blogger.comBlogger55125tag:blogger.com,1999:blog-1629175743855013102.post-15441853822799601102016-03-25T15:22:54.351-07:002016-03-25T15:22:54.351-07:00Like the poster above explained to you that idea s...Like the poster above explained to you that idea supposed a static fixed system with no feedback mechanisms to thwart your efforts at taking in less or expending more. There is significant scientific evidence that the hypothalamus regulates body fat level the way a thermostat controls the temperature in an air conditioned room. It defends a particular body fat level and makes adjustments to metabolism and hunger and satiety that are too powerful to resist long term to ensure that a particular body fat level is maintained. lisaohttps://www.blogger.com/profile/14360700674622979187noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-86842475534252845712010-07-08T11:47:52.332-07:002010-07-08T11:47:52.332-07:00While USDA are busy making the problem worse,
htt...While USDA are busy making the problem worse,<br /><br />http://news.bbc.co.uk/1/hi/health/10545542.stm<br /><br />Meanwhile<br /><br />http://news.bbc.co.uk/1/hi/health/10541190.stm<br /><br />This is seriously scary. it's already finded by Unilever among others, this will just be carte blanche for more pushers of low-nutrition carbs and Omega 6s to purvey their wares in the name of "health"Anonymousnoreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-17142472034966410282010-07-02T23:22:15.379-07:002010-07-02T23:22:15.379-07:00http://www.ncbi.nlm.nih.gov/pubmed/20537171 - That...http://www.ncbi.nlm.nih.gov/pubmed/20537171 - That reminds me of this study found in pub med about nutrient deficiencies. In this study the researcher looked at suggested diet plans from four well-known diets: the South Beach diet, the Atkins for Life diet, the Diet Approaches to Stop Hypertension (DASH) and the Best Life Diet. The Idea was if you followed this diet perfectly would you Percentage of RDI of 27 micro nutrients what you get, all the vitamins and minerals? Basically out of all those diets you would only get between 100% of 12-15 micro-nutrients required out of 27. Not so good, but just popping a muti vitamin is not the answer either, It can help with ones who may be dieting, but choosing to eat foods less unprocessed and more whole you can get most of those requirements by that. Hey ADA recommend 5 servings a day of fruits and vegetables. Screw it GO TO 10 servings of fruits of vegetables a day and mix and match and we should be able to meet allot of those requirements. GREAT POST MUCH AGREED ! :)Nicholas C. Scotthttps://www.blogger.com/profile/13742400069807976432noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-13100557573837581662010-06-30T17:44:08.809-07:002010-06-30T17:44:08.809-07:00So here's an interesting idea. Fat people att...So here's an interesting idea. Fat people attempting to lose weight often fail at very-low-calorie diets. They retain most or all of their fat stores (unless they exercise a LOT... which then kills their lean mass). Normally this is excused away as "starvation mode" because the body is "panicking" at the low caloric intake.<br /><br />But wait. Isn't it more accurate to say that <i>ketosis</i> is "starvation mode"? If our fat stores are meant to take us through lean times, ha ha, then it stands to reason that if we're starving, we need to mobilize those fat stores for energy. It makes no sense to keep the energy stored when faced with few available calories. That's just suicidal. It means you get sluggish, slow down and are mentally less inclined to do anything for yourself. As I'm sure you've noticed about many fat people who have not yet made changes to their diets. I've been there myself.<br /><br />Also, we're smarter, it would seem, on ketosis than out of it--the brain is apparently more efficient. Again, it makes more sense evolutionarily to maintain your energy level and get smarter so that you can find food before you die.<br /><br />Wow.<br /><br />I had already had an idea it wasn't really "starvation mode" that inhibits weight loss on low calories. I have a problem with this myself--I'm working on weight loss, and I often forget to eat. When I track my food and I've had a bad day, I can sometimes find myself under the 1000-calorie mark for intake. I'm in ketosis so it doesn't bother me much until I really get hungry (even then the hunger's different than what occurs from a drop in blood sugar level--a nice change!), but it seems like it is still a problem.<br /><br />Aaand... I <i>have</i> noticed that my weight loss seems to be faster when I'm eating a good 2000 calories or more per day. If my weight loss slows down, and I've been tracking my food, I will usually note that I haven't been eating at that 2000-calorie minimum. Usually it's more like 1600 or less. I had come to wonder whether micronutrient lack might have been the fundamental problem.<br /><br />Your post here is helping put the puzzle together for me. I love it when I might be right. Or am right. Oh well, I have a feeling more research will just confirm this finding.<br /><br />Oh man... JLL, give it up. First off, if you're obese, you want to lose fat, not just "weight." You calorie-restrict to lose the weight and happen to restrict the wrong kinds of calories, the "weight" you lose will be muscle and bone, not fat.<br /><br />I wish people would get over this... I really do... I mean, we don't even burn all the food we eat for fuel! Has someone worked out how much of it goes to fuel and how much to building materials? Has someone worked out whether or when or how those proportions change? Probably not. There's a lot we still don't understand about human metabolism.<br /><br />I <i>wish</i> it were about cutting back 500 calories a day. In my experience, I have to <i>add</i> that 500 to see significant weight loss. Then, on top of that, the fatty acids I'm releasing from my adipose tissue? I'm "eating" those too. I never did burn them for fuel from whenever I ate the original foods. Now I <i>am</i> burning them. So that's extra calories, God only knows how many...<br /><br />...because I'm also in ketosis, which means that when I visit the little girl's room, some of those calories I was supposed to be burning get flushed down the toilet!<br /><br />I love nice, neat answers too. Unfortunately, they seem to be in short supply in this case.<br /><br />Oh, and I bet they'd have an even better outcome in that study if they used retinol (from a natural source, preferably) instead of beta carotene. Just saying.Dana Seilhanhttps://www.blogger.com/profile/11749354913843954242noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-44403874731324643692010-06-25T09:58:02.058-07:002010-06-25T09:58:02.058-07:00The USDA has issued draft dietary guidelines for 2...The USDA has issued draft dietary guidelines for 2010 which recommend further restrictions on consumption of cholesterol and a reduction in saturated fat intake from 10% to 7% of calories.<br /><br />The proposed guidelines will result in reduced the consumption of nutrient dense foods such as liver, eggs and dairy fat and coconut oil and will lead to reduced intake of nutrients essential for good health thereby causing increased obesity and disease.<br /><br />More details on the draft guidelines are given in the action alert from WAPF below:<br /><br />http://www.westonaprice.org/action-alerts/2010-alerts/1949-action-alert-usda-dietary-guidelines-for-2010.htmlAnonymousnoreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-5125808387294797742010-06-24T05:52:55.094-07:002010-06-24T05:52:55.094-07:00Just published in AJCN
Am J Clin Nutr (June 23, 2...Just published in AJCN<br /><br />Am J Clin Nutr (June 23, 2010). doi:10.3945/ajcn.2010.29468<br /><br />Micronutrient quality of weight-loss diets that focus on macronutrients: results from the A TO Z study1,2,3<br />Christopher D Gardner, Soowon Kim, Andrea Bersamin, Mindy Dopler-Nelson, Jennifer Otten, Beibei Oelrich and Rise Cherin<br />ABSTRACT<br /><br />Background: Information on the micronutrient quality of alternative weight-loss diets is limited, despite the significant public health relevance.<br /><br />Objective: Micronutrient intake was compared between overweight or obese women randomly assigned to 4 popular diets that varied primarily in macronutrient distribution.<br /><br />Design: Dietary data were collected from women in the Atkins (n = 73), Zone (n = 73), LEARN (Lifestyle, Exercise, Attitudes, Relationships, Nutrition) (n = 73), and Ornish (n = 72) diet groups by using 3-d, unannounced 24-h recalls at baseline and after 8 wk of instruction. Nutrient intakes were compared between groups at 8 wk and within groups for 8-wk changes in risk of micronutrient inadequacy.<br /><br />Results: At 8 wk, significant differences were observed between groups for all macronutrients and for many micronutrients (P < 0.0001). Energy intake decreased from baseline in all 4 groups but was similar between groups. At 8 wk, a significant proportion of individuals shifted to intakes associated with risk of inadequacy (P < 0.05) in the Atkins group for thiamine, folic acid, vitamin C, iron, and magnesium; in the LEARN group for vitamin E, thiamine, and magnesium; and in the Ornish group for vitamins E and B-12 and zinc. In contrast, for the Zone group, the risk of inadequacy significantly decreased for vitamins A, E, K, and C (P < 0.05), and no significant increases in risk of inadequacy were observed for other micronutrients.<br /><br />Conclusions: Weight-loss diets that focus on macronutrient composition should attend to the overall quality of the diet, including the adequacy of micronutrient intakes. Concerning calorie-restricted diets, there may be a micronutrient advantage to diets providing moderately low carbohydrate amounts and that contain nutrient-dense foods.<br /><br />Received for publication March 4, 2010. Accepted for publication May 23, 2010.Keith Grimaldihttps://www.blogger.com/profile/03340553707811672190noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-23421137244005871932010-06-24T03:34:02.172-07:002010-06-24T03:34:02.172-07:00One of my neighbours has a vintage car which he ra...One of my neighbours has a vintage car which he rallies. It does about 30 mpg (UK gallons) and has the performance of a slug on valium.<br /><br />I have a modern turbodiesel which has more than adequate performance and does well over 60 mpg.<br /><br />The basic principles and components are the same, just more finely tuned. A more efficient burn and lower *internal losses* make the fuel calories produce over twice the output energy. IMO that's what we're trying to address in a human system where we can't simply bolt on a turbocharger or reset a fuel pump.<br /><br />Current diets aren't providing a clean burn and the only way we can get sufficient power out of the system is by overfuelling. Instead of the excess fuel producing smoke and soot it's producing body fat etc.<br /><br />Even a simple mechanical system only does "calories in, calories out" when you take into account all of the processes involved: internal friction, waste heat and sound energy, semi-burned fuel . . .Anonymousnoreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-46474328573416298552010-06-23T19:44:07.996-07:002010-06-23T19:44:07.996-07:00Stephen,
Regarding the subject post, I would like...Stephen,<br /><br />Regarding the subject post, I would like your opinion on the hypothesis that the 40% increase in HDL that occurred after vitamin and mineral supplementation was due to decreased inflammation that reduced catabolism of HDL phospholipids by endothelial lipase (EL).<br /><br />EL is a member of the lipase family that is primarily synthesized by endothelial cells which has been found to play a key role in metabolism of HDL-C. Inflammation causes an increase in EL activity which results in hydrolysis of HDL phospholipids and decreased HDL-C levels, whereas suppressing EL activity enhances concentrations of HDL. Increased EL concentrations are associated with a deteriorated lipoprotein profile, elevated triglyceride concentrations and smaller LDL particle size. EL concentrations are positively correlated with obesity, fasting insulin and markers of inflammation.<br /><br />CoQ10 deficiency causes increased Crp which activates EL and thereby reduces HDL. CoQ10 synthesis requires at least seven vitamins and several minerals. CoQ10 deficiency due to nutrient deficiency is common. In the Li study, the supplements provided included nutrients needed for CoQ10 synthesis and therefore would be expected to result in increased HDL due to lower Crp induced catabolism of HDL by EL.<br /><br />High homocysteine due to B vitamin deficiencies also results in reduced HDL due to increased EL activity.<br /><br />It is my opinion that the increased HDL that occurred following nutrient supplementation was the result of suppression of EL activity due to increased CoQ10 and decreased Hcy (and probably other things too)<br /><br />Weight loss would also be effected by EL, for lipoprotien lipase (LPL) is inversely correlated with EL, so reduced inflammation would cause increased LPL thereby enhancing metabolism of fats and carbohydrates.Anonymousnoreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-14556118239246593872010-06-23T18:46:58.514-07:002010-06-23T18:46:58.514-07:00@Razwell
To me, this article simply show that a l...@Razwell<br /><br />To me, this article simply show that a lack of micronutrient could slowdown BMR and that getting adequate level of micronutrient could make the basal metabolic rate run faster and hence burn more calories. I'm not sure how it's supposed to invalidate the theory. (I've not read the paper tho, not even the abstract, this is only from my reading from Stephan's article.)<br /><br />How would you explain all of the people who lost weight (weight watcher has a lot of success story of 100+lbs lost and maintained) and maintain the lost with basic dieting principle (ie, consume less than you expend) if they are supposedly a total failure?<br /><br />I think the mistake that many people do is to be on either side, whereas it's actualy obvious that in term of energy calorie clearly matters, but that differents calories can have differents metabolic effects.<br /><br />Razwell, are you going to tell me that the answer to the obesity epidemic is to go low-carb? Just like they did in the 70-80' with the low-fat?<br /><br />I'm a calorie matter proponent, and I clearly understand that obesity is complexe. But calorie still do matter, and albeit not 100% precise, it's still hold in most context.<br /><br />Surely, guys like Alan Aragon, Lyle McDonald, Martin Berkhan, who have a lot of (awesome) real world results, and who goes by calories quite a lot, know nothing about nutrition. Sure thing.<br /><br />Why does it have to be black or white? Why can't calorie matter AND metabolic disorder matter too?<br /><br />I don't want to get into the cliché that people are fat because they mean to get there, but people are always looking for a quick fix, for something to put the blame else than on themselve. People don't wanna work, make the effort, change their habits, etc, etc. This is plain human nature. I see this all the time in the gym at which I work. People want the magic supplements, the magic program, the magic diet. They don't like it when I tell them it takes time, patience, and work. Then you've got thing like "fat is not your fault" and people trying to find multiple reason to obesity, such as obesogens, gut microbiota, insulin resistance, etc. This certainly all matter, to some degree, but taking the responsability off the individual isn't going to help him either. <br /><br />Anyway, thanks for your answer Stephan. Your blog is a very intelectualy rich place.François Létourneauhttps://www.blogger.com/profile/10704963616951516321noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-25602423710234859692010-06-23T18:41:39.051-07:002010-06-23T18:41:39.051-07:00This comment has been removed by the author.François Létourneauhttps://www.blogger.com/profile/10704963616951516321noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-35193707188627954932010-06-23T06:36:56.761-07:002010-06-23T06:36:56.761-07:00Also, you are right Stephan about common calory th...Also, you are right Stephan about common calory theory proponents farcical arguments. <br /><br /><br />Their "basic thermodynaimcs" argument mixes up E=MC^2 in a way Einstein would not forgive. Humans are not closed systems, and no caloric energy is being turned into matter in a human.<br /><br />A calorie is a unit of heat used in physics. It has has everything to do with the energy liberated from burning a substance. It hs very, very little to do with biological processes- which do not involve combustion. The Caloric Model is a 60 year old rough analogy. It considers food for its combustive energy and completely ignores what actually happens to food molecules inside the body . Overeating is bad, of course. But the phenomenon of obesity is much. much more complex than calorie theory proponents will admit........<br /><br />The main faults of the Caloric Model is that it attempts to posit a closed system where none exists, it grossly oversimplifies,and it does not attempt to explain many, many observations. A good way to get a theory killed is by ignoring contradictory evidence.<br /><br /><br />The question is not whether the Laws of Thermodynamics apply to humans. The question is whether we can infer from these laws the behavior of mammalian fat cells. <br /><br />Thermodynamics requires that energy is conserved in a system , and it is. But whether lipids are stuffed into fat cells , processed to extract chemical energy , or simply eliminated as waste is not predicted by those laws. Those things are governed by other variables. <br /><br /> <br />Anyone who invokes the Laws of Thermodynamics in an obesity discussion has absolutely no idea what they are talking about. They are taking the laws completely out of context and attempting to oversimplify a very complex phenomenon. Funny thing. Actual biologists aren't much into oversimplifying a physiological phenomenon. Mostly where it happens is in the diet industry where proponents have a financial stake in the conclusions drawn. <br /><br /><br />The Laws of Thermodynamics are not violated by acknowledging that in the non closed system that which is a mammal , how fat cells behave is governed by chemical signals - from hormones and food substances- and by states of disease and genetics.<br /><br />To lose fat , fat cells must be made to disgorge themselves of their excess lipid molecules and allow the body to eliminate them as waste. Saying this does not violate the Laws of Thermodynamics.<br /><br /><br />GREAT blog by the way, Stephan. I am very happy this info is getting out there. <br /><br />I encourage EVERYONE on here to forward this article to ALL calorie theory proponents you know , including all these popular fat loss gurus with massive followings ( who shall remain nameless) on the internet who think they are experts. They're not. They push dieting and diets do NOT work. Anyone who understands anything about obesity would NEVER push dieting. The fact these gurus do, should tell you all you need to know about them.<br /><br />Science just does not know enough yet about the regulation of mammalian fat cells for such cocksuredness from proponents of the failed caloric model of obesity.Anonymousnoreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-10067547157290028432010-06-23T06:05:41.083-07:002010-06-23T06:05:41.083-07:00Stephan
Traditional Asian diets definitley consis...Stephan<br /><br />Traditional Asian diets definitley consist of various organ meats, land meats, seafood like octopus, various insects and bugs , snakes, (yes, you read that right), rats in some areas. You will find some of the strangest animal foods ever in Asia. It is unreal.<br /><br />Zimmerman's and Bourdain's shows cearly show how animal food is a major part of all Asian diets, despite vegan propaganda. Anyone who has been there already knows this.Anonymousnoreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-1868713898475761642010-06-22T23:57:39.061-07:002010-06-22T23:57:39.061-07:00The calorie equation fits OKish probably for the m...The calorie equation fits OKish probably for the majority of food eaten - but it's not a perfect sum and therein is the problem. But really why should it be thought to be accurate? Calorie is a measure of heat - all things "contain" calories but a 5000 cal/day diet of grass won't make you fat (just sick), but 5,000 cals of burgers+buns probably would.Keith Grimaldihttps://www.blogger.com/profile/03340553707811672190noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-72499952168400306802010-06-22T16:58:48.645-07:002010-06-22T16:58:48.645-07:00Beautifully said.Beautifully said.Helenhttps://www.blogger.com/profile/14504810823521044641noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-82121961742064721982010-06-22T13:54:11.280-07:002010-06-22T13:54:11.280-07:00I wanted to follow up on my previous comment with ...I wanted to follow up on my previous comment with an example to illustrate what I'm talking about. Leptin receptor mutated rats (Zucker rats) are extremely fat. It's literally impossible to make them lean by reducing their food intake. They will die of starvation with large fat depots, because they break down their lean mass to spare their fat. The same is true (to varying degrees) in a number of other obesity models, including ovariectomized rats and diet-induced obese rats. <br /><br />The same thing happens in humans as well. If you take an obese person and reduce their calorie intake (while keeping diet composition and lifestyle constant), they will lose both fat and lean mass. However, their body goes into "conservation mode" as leptin drops, reducing energy expenditure, increasing appetite, etc. Therefore, in the long term, the body fights fat loss using a number of strategies, so telling a person to eat less or exercise more is virtually useless in practice (in the long term). <br /><br />Yes, you can lose fat simply by restricting calories if you have an iron will and you can starve yourself enough. But you will also lose lean mass, and be hungry and miserable, because your body will think you're starving even if you still have a high fat mass. Furthermore, 99% of people lack the mental resolve to fight their hypothalamus indefinitely. None of this contradicts the laws of thermodynamics, but it does contradict the oversimplified interpretation of thermodynamics that's most commonly stated by diet authorities and the media.Stephan Guyenethttps://www.blogger.com/profile/09218114625524777250noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-18265962317634825062010-06-22T10:46:28.736-07:002010-06-22T10:46:28.736-07:00Hi Francois,
Yes, that formula is correct as long...Hi Francois,<br /><br />Yes, that formula is correct as long as you assume a constant fat and lean mass. The "calories in, calories out" question is nothing more than a semantic issue. I'm not questioning the thermodynamics of the human body, only the oversimplified version that has trickled down into the popular consciousness, that people use to justify the "gluttony and sloth" explanation of obesity.Stephan Guyenethttps://www.blogger.com/profile/09218114625524777250noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-76300383043479746882010-06-22T06:20:37.267-07:002010-06-22T06:20:37.267-07:00Hi Stephan.
Thanks for the answer.
Am I wrong or...Hi Stephan.<br /><br />Thanks for the answer.<br /><br />Am I wrong or the calorie in = calorie out is actualy<br /><br />Calorie in = RMR (REE) + TEF + Physical activity (which could be divide in non-spontaneous and spontaneous)<br /><br />If you influence REE, you simply influence the calorie out of the equation. Or am I wrong?François Létourneauhttps://www.blogger.com/profile/10704963616951516321noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-35292316141848228662010-06-22T06:09:48.631-07:002010-06-22T06:09:48.631-07:00This comment has been removed by the author.François Létourneauhttps://www.blogger.com/profile/10704963616951516321noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-59959582421514568612010-06-21T18:40:39.501-07:002010-06-21T18:40:39.501-07:00You might be interested in this link.
http://www....You might be interested in this link.<br /><br />http://www.nutritionandmetabolism.com/content/3/1/35<br /><br />"Vitamin C is a cofactor in the biosynthesis of carnitine, a molecule required for the oxidation of fatty acids. A reduction in the ability to oxidize fat may contribute to the reported inverse relationship between vitamin C status and adiposity."stanhttps://www.blogger.com/profile/12013025815871749498noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-3298858030442165272010-06-21T10:01:11.817-07:002010-06-21T10:01:11.817-07:00Hi Francois,
The "calories in, calories out&...Hi Francois,<br /><br />The "calories in, calories out" hypothesis, as commonly defined, does not account for changes in resting energy expenditure. It states that voluntary calorie intake and physical activity behaviors are the determinants of fat mass. In other words, fat mass is under conscious control. It's partially true, but not particularly relevant in practice due to homeostatic feedback mechanisms on fat mass.Stephan Guyenethttps://www.blogger.com/profile/09218114625524777250noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-26222688774719673922010-06-21T09:56:34.996-07:002010-06-21T09:56:34.996-07:00@ Trinkwasser
"Keshan disease" (selenium...@ Trinkwasser<br /><a href="http://pubs.acs.org/cen/80th/print/selenium.html" rel="nofollow">"Keshan disease" (selenium deficiency related) was first reported in Keshan Heilongjiang province .</a> <br />Harbin Medical University, Harbin, Heilongjiang province conducted the study <i>Effects of multivitamin and mineral supplementation on adiposity, energy expenditure and lipid profiles.</i> it's likely the study participants were local residents.)TedHutchinsonhttps://www.blogger.com/profile/13140097526458431747noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-60131767303409060392010-06-21T08:32:11.802-07:002010-06-21T08:32:11.802-07:00I had a sudden thought: parts of China are serious...I had a sudden thought: parts of China are seriously selenium deficient. Other parts have an excess. Would be interesting to know which part this population inhabited, if the supplements are simply replacing the missing selenium (possibly also iodine) this might be readjusting thyroid function in a beneficial direction.Anonymousnoreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-26116856317734674072010-06-21T08:02:02.873-07:002010-06-21T08:02:02.873-07:00Stephan
I'm not sure how this is supposed to ...Stephan<br /><br />I'm not sure how this is supposed to invalidate the calorie in = calorie out theory.<br /><br />You said they lost more weight because of an increase in REE. <br /><br />So it simply influenced the calorie out part of the equation, I don't see how it negate the whole thing.<br /><br />Just like with the TEF of protein. It's not that the equation does not hold, you simply influence more the calorie out part of it.François Létourneauhttps://www.blogger.com/profile/10704963616951516321noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-90131608630827201192010-06-20T21:10:27.983-07:002010-06-20T21:10:27.983-07:00Jack C,
That squares with what I learned doing fo...Jack C,<br /><br />That squares with what I learned doing food security work in the United States. As income goes up, the quality of food eaten goes up. It's not rocket science. Nor are the food choices just a matter of "ignorance," as many would like to believe of those plagued with both obesity and poverty. <br /><br />The areas in which there was the most hunger, according to our research (and there was widespread, intermittent, actual hunger - involuntarily skipping meals, losing weight, and eating less due to lack of food - when you can't even afford the cheap food), also had the most obesity. I'm sure that, along with micronutrient status and fat-producing inputs (HFCS, omega-6), the body's reaction to intermittent caloric shortages (Mark's Daily Apple explained it well recently in a post about carbohydrate refeeding on a low-carb diet), tend to make it hang on to weight in spite of lower calories.Helenhttps://www.blogger.com/profile/14504810823521044641noreply@blogger.comtag:blogger.com,1999:blog-1629175743855013102.post-70129062719131657042010-06-20T12:55:33.759-07:002010-06-20T12:55:33.759-07:00Bryan,
Insight into just how lame the "calor...Bryan,<br /><br />Insight into just how lame the "calories in, calories out" theory is can be gleaned from the data from the Epic-Heidelberg study on the correlation between vitamin K2 intake and prostate cancer.<br /><br />In the study, 11,000 men were followed for 8 years and the correlation between K2 intake and prostate cancer was determined based on food frequency analysis.<br /><br />Men were ranked by quartiles of vitamin K2 intake, and because cheese is rich in K2, cheese consumption among those in the quartile of K2 intake was four times that of the quartile with the lowest K2 intake.<br /><br />The percentage of those with University degrees was 44% in the highest Q of K2 intake compared to 29% with the lowest K2 intake, suggesting that those that ate a lot of cheese had greater wealth.<br /><br />Those in the highest quartile of K2 intake had a BMI and 26.8 and a daily caloric intake of 2730. By comparison, those in the lowest quartile of K2 intake had a higher BMI, 27.1, but a caloric intake of of 1724 kcal/day. Other factors such as exercise, smoking and alcohol consumption were about the same for the two groups.<br /><br />The low K2 group, the less wealthy, ate cheaper food, more omega 6, more carbohydrates, less protein and less fat than the wealthier group and were fatter even though they ate 35% fewer calories. "Calories in, calories out" does not fit the data at all does it.Anonymousnoreply@blogger.com